Postdoctoral fellowships are available to study the molecular and cellular bases of neuronal dysfunction using the pediatric disorders, spinal muscular atrophy (SMA) and Glut1 deficiency syndrome (Glut1 DS) as disease paradigms. SMA is the most common genetic cause of childhood mortality. Glut1 DS is a significant cause of pediatric epilepsies. Current projects involve using model mice (J. Clin. Invest. 130: 1271;Neuron, 111: 1-17; J. Clin. Invest.124: 785; Nat. Commun. 8:14152) to 1) Define the cellular site(s) of action of the SMN and Glut1 proteins in causing neuronal degeneration, 2) Determine molecular pathways that link reduced SMN or Glut1 protein to neuronal loss or dysfunction and 3) Develop novel treatments for SMA, Glut1 DS and other neurodegenerative diseases. Applicants must have a published track record in molecular genetics. Expertise in mouse neurobiology, RNA biology and bioinformatics is highly desirable. In addition candidates must be team players, demonstrate initiative and communicate effectively. The salary for the position is highly competitive and the academic research environment is internationally recognized as being among the best.
A letter describing your research interests, CV and two letters of reference will constitute an application. Send information to Umrao R. Monani, PhD, 630 W. 168th St., Rm. 5-422, Columbia University Medical Center, New York, NY 10032 or to email@example.com [Subject line: Attn: Dr. Umrao R. Monani –PF].
Columbia University is an Equal Opportunity Employer / Disability / Veteran
Pay Transparency Disclosure
The salary of the finalist selected for this role will be set based on a variety of factors, including but not limited to departmental budgets, qualifications, experience, education, licenses, specialty, and training. The above hiring range represents the University's good faith and reasonable estimate of the range of possible compensation at the time of posting.
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